Here’s a summary of the podcast episode Dolorology (PAIN) with Dr. Rachel Zoffness (from the series Ologies with Alie Ward) featuring pain-psychologist Rachel Zoffness, including its key ideas and implications.
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In the episode, Dr. Zoffness asks: What is pain? Where does it come from? And how can we hurt less?
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She explains that pain is not simply a signal of damage in the body — it’s a complex brain-based construction, shaped by biology, psychology and social factors.
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The term “dolorology” (from Latin dolor = pain) is used to describe the study of pain: its nature, experience, and management.
Key Concepts & Insights
Pain is constructed in the brain rather than being purely a literal measure of tissue damage. For example, someone may perceive pain (like phantom limb pain) even when the limb is absent — indicating the brain’s “danger alarm” is at work.
Pain is Biopsychosocial:
- Bio = tissue status, genetics, injury, sleep, exercise, physical health
- Psycho = attention, emotion, cognition, beliefs about pain
- Social = context, support, work environment, stress, trauma
- Dr. Zoffness emphasizes that all three domains matter in how pain is experienced and maintained
“Pain recipes” and amplifiers: She describes that pain is often the result of a “recipe” of factors, not just one thing. For example: stress, sleep loss, injury, anxiety, social isolation might all amplify the “pain dial” in the brain.
Context, attention, and emotion matter:
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If you’re in a painful injury but surrounded by distractions, positive context, or focusing elsewhere, pain may be less intense.
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If you’re immobilized, anxious, depressed, isolated, or ruminating — pain gets amplified.
She gives the “tale of two nails” example: one person experienced intense pain for a nail through a boot, another person had a nail embedded in his face/brain for days with much less pain. The difference: context, brain interpretation, expectation.
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Chronic pain = sensitization: When pain lasts for weeks/months, the nervous system becomes more sensitive — smaller triggers lead to larger pain responses. This is central to chronic pain conditions.
Treatment implications:
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Understanding pain neuroscience is key (“pain education”) so patients and clinicians stop treating pain as purely structural/damage-based.
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Cognitive-behavioural and mindfulness-based strategies help patients hack their pain recipe — reduce the amplifiers.
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Simply pursuing structural fixes (scans, surgeries) may miss the bigger picture of the pain system.
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Practical Takeaways
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If you’re working with someone (or yourself) with pain: ask about sleep, mood, stress, attention, social environment, expectations — not just “what’s wrong physically?”
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Use language that helps someone understand pain is real, but also modifiable — the brain can turn things down.
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Encourage activities/contexts that shift attention away from pain, increase positive/social engagement, improve movement and function rather than only focusing on reducing pain intensity.
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Be cautious: scans/imaging may show “damage,” but that doesn’t always correlate with pain severity or future risk.
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For clinicians: include pain neuroscience education in treatment plans; consider behavioural/psychological interventions earlier rather than only after structural treatments fail.
- Stress and anxiety change pain volume – be calm and relaxed
- Mood and emotions change pain volume – be in joyous happy circumstances
- Attention changes pain volume – focus on other things
